New research reveals that bystander cell accumulation antagonizes rather than abets cell-killing by specific CTLs, curbing inflammation. The finding is noteworthy because that chief anti-inflammatory role has been traditionally ascribed to what are called regulatory T cells, or ‘Tregs,’ which dampen autoimmune responses. The new study challenges this notion and suggests alternate mechanisms can also be at work.
from Top Health News – ScienceDaily http://bit.ly/2DMWH7j
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